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Hypoxic Ischemic Encephalopathy - Oren Zarif - Hypoxic Ischemic Encephalopathy

Although the causes and prognosis of hypoxic ischemic encephalopathies are not yet fully understood, there is some evidence of the disorder. In neonates, MRI is recommended for all cases of encephalopathy, seizures, or head trauma to assess the extent of brain damage and determine the diagnosis and prognosis. In addition, head ultrasound can help diagnose hemorrhage and ventriculomegaly in infants, both of which are normal following a hypoxic ischemic event. CT is not commonly used in newborns due to the high radiation doses.

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HIE is classified into three stages based on the type of brain injury it causes. The first stage is mild or no brain injury, while the second stage is moderate to severe. Although the severity of this form of HIE is often unclear, treatments are available to help patients recover quickly. In addition to TH, there are also a number of therapies for children suffering from moderate to severe HIE. A specialized care team can help you decide which treatment is right for your child.

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Once the diagnosis of HIE is made, therapeutic hypothermia is usually administered. Early EEG and MRI findings are prognostic for treatment. However, even mild HIE is associated with low Apgar scores and severe acidosis. Treatment should target both acute and subacute injury mechanisms to prevent further damage. This disease is an important cause of childhood developmental disability, and it is the most common form of ischemic encephalopathy in infants.

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The best treatment for hypoxic ischemic encephalopathic (HIE) is to prevent asphyxia during pregnancy. Awareness of risk factors can help parents prepare for the occurrence of a child with HIE. The terminology is specific but similar to other brain disorders. Some of the most common terms related to this disease are brain lesion, brain malformation, and brain defect. Learning about the causes of HIE will help you better understand what causes the symptoms of cerebral palsy and how to prevent them.

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Hypoxic ischemic encephalopathy is the result of limited oxygen and blood flow to the brain during birth. The condition can be mild or severe, depending on the length of time the brain is without oxygen. While some children will suffer only mild symptoms, some may have permanent disability. A comprehensive evaluation will determine the causes of HIE and the treatment options. In severe cases, the condition can result in death or disability.

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Preterm cerebral hypoxic ischemia may occur in very rare cases because of a recognized sentinel event. Because of the difficulty of distinguishing these two pathologies, the definition of HIE in preterm infants remains a complex problem. Few studies have been published specifically on preterm HIE. These studies also lack uniform inclusion criteria. As a result, the diagnosis of HIE remains controversial, and there are many underlying causes of the disorder.

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Perinatal hypoxia can be devastating for the infant's brain. It can result in mild behavioural deficits to severe neurologic sequelae, including cerebral palsy and seizures. Although no specific therapeutic intervention is available to treat hypoxia, future neuroprotective therapies are anticipated to improve neurological outcomes. One promising therapy for hypoxic ischemic encephalopathy is hypothermia. The use of neonatal rat models may improve the treatment of HIE.

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Early recognition of the encephalopathy can help exclude other causes and facilitate the patient's prognosis. Early treatment is usually supportive and does not require surgery. However, if treatment is delayed, the symptoms may worsen. In such cases, the patient should be monitored carefully and undergo appropriate neuroprotective measures. There are other causes of encephalopathy, including the presence of tumors and anemia.

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