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What to Expect After a Cerebral Infarction - Oren Zarif - Cerebral Infarction


After cerebral infarction, a person's brain begins to show changes. The gray-white junction in the cerebral cortex may disappear, and the infarcted region begins to separate from the surrounding, viable brain tissue. Hemorrhagic infarction develops as the blood vessel walls become weakened. It may take weeks for the entire area of brain to recover from cerebral infarction. Here is what to expect during the recovery period.

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There are many causes of cerebral infarction. A recent study involving a large number of young people showed that cardiovascular risk factors are associated with a greater risk for cerebral infarction. This is not always the case. Some vascular risk factors, such as diabetes and high blood pressure, can lead to an increased risk of cerebral infarction. While these are just a few possible causes, the fact that stroke is a common medical condition is worth considering.

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Another possible cause of infarction is hypotension. Hypotension causes the blood pressure to rise above normal levels. Hypertension and hypotension can also cause cerebral infarction. The areas of infarction are usually discolored and expanded. A patient with cerebral infarction is likely to have a variety of symptoms, including a loss of sensation in the opposite side of the body. They may also experience decreased eye movement. Speech may be affected by an infarction in the left side of the brain. Reflexes may also be aggravated.

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The most common cause of cerebral infarction is arterial-to-artery embolism. Watershed infarcts may occur less frequently because of the presence of hemodynamic instability. An embolic infarct associated with a plaque tends to involve the middle or posterior cerebral artery territory and is wedge-shaped on neuroimaging studies. It may also be caused by a collateral anastoma.

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An initial workup for cerebral ischemia includes basic labs, such as a complete blood count, coagulation factors, and EKG. In addition, a stat non-contrast head CT is recommended to rule out a mass lesion or hemorrhage. Vascular imaging may be useful in identifying the cause of acute stroke. Acute large vessel occlusion is often obvious on CT images.

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There are many treatments available for patients with acute cerebral ischaemia, including thrombolytics and mechanical removal of the blockage. A thrombolytic drug called tPA (timolol) breaks up the clot, allowing blood to flow through the brain. This treatment is an option for patients with a high risk of dying from a stroke. It can also prevent the sudden onset of posterior fossa compression syndrome.

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Another type of cerebral infarction is ischemic stroke. This type of stroke is caused by problems with the blood vessels that supply the brain. This results in a lack of oxygen and essential nutrients for brain cells. Some areas of the brain are affected and die as a result. However, in many cases, cerebral infarction is caused by other factors. If you suspect that you have cerebral ischemia, consult a doctor right away.

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Infarction of the brain is a common result of a thrombotic hemorrhage. There are many possible causes, including pulmonary embolism. However, you should never ignore the risk factors if you have a history of cerebral infarction. You may want to consider a referral to a cardiologist if you suspect that you have an ischemic brain disease. A physician will be able to determine if you have cerebral infarction and prescribe a suitable treatment.

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The risks of recurrent stroke are particularly high in people with atrial fibrillation. In addition, immediate anticoagulation of acute cardioembolic infarct is associated with a higher risk for hemorrhagic transformation. Even if your acute infarct is not large, early anticoagulation may be appropriate. A patient with uncontrolled hypertension, atrial fibrillation, or both should be consulted immediately.

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As with all ischemic events, stroke occurs in one percent to three percent of people with acute myocardial infarction. However, this percentage is higher in anterior wall infarcts (AMI) compared to inferior wall infarctions. It is also believed that most strokes after an AMI are atherothrombotic or embolic, but most are due to hemodynamic compromise. Most strokes are acute and occur within the first weeks of infarct, but the risk is persistent. Some research indicates that approximately 40% of patients with anterior wall MI have left ventricular mural thrombosis. However, this is not the case with inferior wall MI.

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