Treatment For Wallenberg Syndrome - Oren Zarif - Wallenberg Syndrome

Treatment for Wallenberg syndrome varies greatly, depending on the location of the stroke in the brainstem and the severity of the damage to the underlying structures. People with minor damage can recover in a few weeks, while people with severe damage may require years of treatment and may even have permanent disabilities. Discuss your long-term outlook with your doctor, and be sure to follow the treatment plan your doctor prescribes. Although the disease is often treatable, there are a few important precautions you should take.

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If you are experiencing any of the symptoms listed above, you may be suffering from the Wallenberg syndrome. This syndrome is often also known as lateral medullary syndrome or posterior inferior cerebellar artery syndrome. In either case, it is the result of a blockage of the posterior inferior cerebellar artery, a branch of the cerebral arteries that supplies the brain. When this happens, blood flow to the brain is blocked, causing an infarct to form in the lateral medulla. The infarcted area is characterized by a numbness and a sharp pain in the lower extremities and a loss of consciousness.

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The prognosis of Wallenberg syndrome depends on the severity of the infarct, but it is still a serious condition. Regardless of the severity of your stroke, you need early physical therapy and occupational therapy to ensure you recover the maximum amount of function possible.

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Occupational therapy is also important for patients with Wallenberg syndrome, and doctors will help you find the best rehabilitation program for your specific needs. And if you're worried about your own health, you should consider speaking with a physician about possible treatments.

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Treatment for Wallenberg syndrome varies greatly. While stroke is the most common underlying cause of the syndrome, other conditions like multiple sclerosis, cerebrovascular disease, or tumors of the central nervous system can also cause the condition. Fortunately, the American Stroke Association is dedicated to the prevention, treatment, and prevention of stroke. The National Institutes of Health conducts research about the brain and nervous system, including Wallenberg syndrome.

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Early diagnosis is the key to preventing further complications and improving quality of life for patients with Wallenberg syndrome. Diagnosis is based on a variety of criteria, including the presence of a symptomatic or chronic condition that has already affected the brain. In the event that your symptoms improve, your physician may recommend a surgical procedure to repair the problem. If your doctor suspects a degeneration of the posterior inferior cerebellar artery, you may need a spinal angioplasty.

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In addition to surgical treatment, a therapist may also recommend a variety of speech therapy and neuromuscular electrical stimulation. These approaches vary widely depending on the specific deficits present in the patient. While NMES and speech therapy are the most common treatments for Wallenberg syndrome, the use of VitalStim by a speech and language pathologist may be an option for certain patients. Occupational therapists often administer VitalStim and recurrent neuromuscular stimulation.

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The neurological cause of WS is the loss of neuronal connectivity between the NA and the submental gyrus. This disrupts the function of premotor neurons that control swallowing. Hence, LMI may result in bilateral swallowing muscle dysfunction in patients with WS. In addition, the loss of neuronal connections between the two swallowing centers may also contribute to the development of dysphagia. While the exact mechanism for the loss of this neuronal connectivity is unknown, the underlying causes of the dysphagia are likely to include LMI and a lack of the underlying neural pathways in the brain.

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The major clinical findings associated with swallowing dysfunction in patients with WS are summarized in Table 1. Compared to patients with hemispheric stroke, dysphagia and swallowing disorder are more prominent in WS. Laryngeal elevation is delayed in WS patients and is accompanied by difficulty in controlling bolus. Patients with hemispheric stroke also have difficulty controlling the oral phase of swallowing.