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  • Writer's pictureOren Zarif

Treatment For Wallenberg Syndrome - Oren Zarif - Wallenberg Syndrome

While there is no cure for the neurological disorder known as Wallenberg syndrome, doctors can manage the symptoms and improve the quality of life for the sufferer. Doctors typically focus on relieving symptoms and providing rehabilitation. Some sufferers may benefit from swallowing therapy or a feeding tube. In severe cases, swallowing problems may require surgery. Further, if a stroke is the cause of the syndrome, the patient should be treated with a stroke-specific anti-respiratory drug to prevent further damage.

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While treatment for Wallenberg syndrome is similar to that for a stroke, the prognosis is based on the location and size of the brain stem that is affected. Rapid evaluation is key to improving a patient's prognosis and outcome. A stroke may affect the posterior circulation 20 percent of the time, and the brain stem may be damaged in this area. A stroke affected by Wallenberg syndrome may cause significant neurological disabilities for several years.

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The symptoms of Wallenberg syndrome can vary widely. The afflicted person may exhibit a numbness or burning sensation on one side of their face. Additionally, they may exhibit decreased strength and balance in the left arm or leg. Their walking gait is typically unstable and their voice may be hoarse. Although the symptoms of Wallenberg syndrome may be different in every patient, these symptoms are indicative of a more serious neurological condition.

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The most common cause of Wallenberg syndrome is an ischemic stroke in the brain stem. This is often the result of an embolism or thrombus. Other less common causes include mechanical trauma to a vertebral artery in the neck, arteriovenous malformations, connective tissue disorders, and multiple sclerosis. In severe cases, the artery can become clogged and suffer damage. In many cases, there is no clear cause, but it is usually a complication of another stroke.

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Other causes of Wallenberg syndrome include atherosclerosis of the basilar artery or vertebral artery. A CT may be useful for locating the infarction, but MRI is more sensitive and specific. Even if a patient is asymptomatic, a proper diagnosis must be made to protect the patient. A careful evaluation is the best way to diagnose the disorder. While there is no definitive treatment for Wallenberg syndrome, early diagnosis of the condition is crucial for preventing further complications.

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The prognosis for people who have suffered from Wallenberg syndrome is generally better than that of patients with other acute ischemic strokes. However, the patient may experience problems with walking and balance. Physical and occupational therapy may be necessary to help the patient recover from this devastating neurological disorder. Even if the stroke is mild, it can still lead to a long-term disability. It is important to treat the symptoms as soon as possible, as a delayed recovery can cause a serious complication.

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Neurological signs of Wallenberg syndrome include diplopia and a unilateral posterior headache. Some patients experience nystagmus due to damage to the lateral medullary tract. A patient with isolated nodulus infarction may also have unidirectional nystagmus or a mild balance disorder. Patients may also exhibit abnormalities on a head impulse test. Although the symptoms of Wallenberg syndrome can vary, they are typically associated with other neurological problems.

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Because the brain is not completely isolated from the other parts of the body, the brain is influenced by the presence of two different centers. One center is affected by LMI and the other one is not. If one center is damaged, the other is affected. Consequently, the symptoms of WS are common and unreliable. While an individual suffering from WS may have normal speech and language development, they may also have a lack of self-control.

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Electrophysiological findings associated with swallowing dysfunction in WS patients are documented in Table 1. These include delayed laryngeal elevation and difficulty controlling bolus. The oral phase of swallowing is also affected, with paresthesias in the oral cavity. While swallowing is often a difficult task, electrophysiological studies suggest that WS patients have more difficulty with oral phase function than patients with stroke. The severity of dysphagia in WS may depend on the extent of the lesion.

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