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Thalamic Infarction - Oren Zarif - Thalamic Stroke
Thalamic infarction represents a characteristic stroke syndrome with clinical and radiological characteristics that is likely underdiagnosed. Other lesions may be due to deep cerebral venous thrombosis, with characteristic radiological and neuropsychological features. Thalamic lesions should be considered in the differential diagnosis of intracranial artery occlusion. The diagnosis of thalamic infarction is complicated by several factors.
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Previous studies have described the anatomy of the thalamic artery and its vascular supply, but there is little information about its long-term outcome. A retrospective study conducted in China evaluated the etiologies, risk factors, and clinical outcomes of thalamic infarcts in patients with different ages. The patients were found to have a greater risk for thalamic infarction in men than in women, with most surviving with only mild sensory impairment. Those with a longer OTD, however, were more likely to experience a fatal stroke.
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The symptoms of thalamic stroke depend on the location of the infarct and the type of vascular disease. The most common type of thalamic hematoma involves the thalamogeniculate artery. It is often accompanied by abnormal involuntary movements. If you experience these symptoms, it is time to seek immediate medical attention. It is important to note that thalamic stroke is a complex, multifactorial, multisystem disorder.
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Left hemisphere lesions involve decreased verbal output, impaired comprehension, and hypophonic speech. They may also cause semantic and phonetic errors. Left thalamic aphasia occurs with impaired verbal output, though repetition and acalculia are well preserved. It may affect behavior. This disorder is rare but potentially life-threatening. You should seek medical attention as soon as you suspect a thalamic stroke.
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Treatment after a thalamic stroke focuses on recovery and reducing the risk of future strokes. Rehabilitation focuses on physical therapy, occupational health, and speech therapy. The outlook after a thalamic stroke varies greatly from person to person, but recovery is possible. The first step is understanding how the brain responds to rehab. Once a stroke survivor is aware of the extent of the injury and its severity, rehabilitation can begin.
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Differential thalamic contributions to mnestic-processing is another factor that contributes to its complexity. Some research suggests that the distribution of stroke in the right thalamus is related to the degree of lateralization of the thalamus. Therefore, an MRI may help the doctor diagnose a thalamic stroke faster. This diagnosis is also important in terms of treatment after a thalamic infarction.
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More left-sided thalamic stroke patients are treated for symptoms related to a dominant hemispheric stroke than those with right-sided thalamic infarction. The latter group suffers from left-sided lesions. The lesion maps may show the asymmetry between anteromedian and thalamic vascular territories. This is important since these two areas are critical to language and other visuo-spatial neurocognitive functions.
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Tremor following a thalamic infarction has several causes. A thalamic infarction disrupts important connections and fibres in the thalamus and can cause acute movement disorders. However, due to cerebral plasticity, a delayed onset of symptoms is possible. In some cases, a thalamic infarct in the hand region may cause the hand to regain a smooth coordinated movement, or abnormal involuntary movements.
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Although a thalamic stroke has distinct clinical features, the underlying cause of the condition is unclear. In general, stroke units rely on clinical findings to provide neurocognitive information. In the majority of cases, however, patients are diagnosed only after neuroimaging studies have been performed. Therefore, the symptoms and prognosis of thalamic stroke are based on the clinical findings of treating physicians. The road to recovery is not easy, but recent advances in stroke care have helped many people regain a healthy life.
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