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  • Writer's pictureOren Zarif

Thalamic Infarct and Rehabilitation - Oren Zarif - Thalamic Stroke


When a thalamic lesion is involved, language disturbances can occur. This type of aphasia is characterized by impaired fluency and comprehension of speech, as well as reduced verbal output. Phonological and semantic errors may also be present, but repetition and acalculia are well preserved. A thalamic lesion can also lead to atypical behavior, including aggression.

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Two other vascular syndromes can result from a thalamic infarct. Acute cerebral ischemia may occur in the thalamus due to a pulmonary embolism. Symptoms of thalamic stroke can be difficult to recognize, as the diagnosis is based on a mix of pathology. Although a thalamic lesion can cause a variety of symptoms, it is most often the result of a single-vessel occlusion. The artery that originates in the posterior horn of the brain is commonly a pulmonary artery.

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MRI shows a small acute infarct in the tuberothalamic artery territory and adjacent central midbrain. The thalamic lesion can be seen on both sagittal and axial T2 FLAIR images. The patient was also found to have a basilar embolus. Moreover, a ventriculoperitoneal shunt was performed. These scans are helpful in diagnosing thalamic stroke.

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MRI and functional assessments of spinothalamic structures were useful in identifying patients at increased risk for thalamic pain after a thalamic stroke. While the results of previous anatomical studies have demonstrated the role of the spinothalamic system in central pain, no studies have attempted to combine the two in a single study. This study combined quantitative sensory examination with physiological recordings of spinothalamic evoked potentials in 42 thalamic stroke patients with or without central pain.

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Despite the significant limitations of thalamic stroke, rehabilitation is a vital part of treatment. Rehabilitation involves learning how to compensate for compromised abilities. Recovery is often facilitated by integrating these skills into daily life. Rehabilitation can improve these secondary effects and increase the chances of a full recovery. This method was developed to measure the impact of thalamic stroke rehabilitation on neurological functioning. It is particularly effective when rehabilitation is performed after the stroke.

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Spinothalamic dysfunction was first reported in the late 1980s and has been replicated many times since. In a study performed by Garcia-Larrea et al. (2010), thalamic lesions were identified, and then projected onto axial sections of the Morel atlas, confirming that the CPSP was caused by a thalamic lesion. In the present study, CPSP was the most important anatomical criterion that predicts pain after a thalamic stroke.

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During a thalamic stroke, the region of the arteries that supply the thalamus has an abnormality. When an infarcted portion of the thalamus is impacted, the resulting damage can occur in a variety of ways, depending on the location of the stroke. Regardless of the location, a stroke is a traumatic event in the brain. Rehabilitation is a crucial part of recovery.

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During the first two weeks after a thalamic stroke, patients often experience varying degrees of pain. Patients who develop pain after a thalamic stroke have MRI lesions in the anterior pulvinar region. A more comprehensive thalamic stroke diagnosis is essential. By identifying the individuals at high risk, treatment can be initiated. If you have any of these characteristics, you may be at risk.

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Using the statistical methods described in previous studies, a model combining pain and thalamic pain was developed that had 85% predictive accuracy for thalamic pain. However, this model did not account for the presence of paresthesia, a condition that is more common in patients with thalamic stroke. This study is still ongoing, but it will help researchers understand this important neurological condition. Its effects on brain function are essential for determining what causes a thalamic stroke.

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Anterior pulvinar nucleus (APN) lesions in thalamic patients may play a critical role in the development of pain after a thalamic stroke. These lesions, a part of the spinothalamic system, are associated with pain and lemniscal dysfunction. Furthermore, a central pulvinar lesion is a significant risk factor for thalamic pain.

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