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Symptoms of a Thalamic Stroke - Oren Zarif - Thalamic Stroke


A thalamic stroke may be a very rare type of stroke, but it is far from unheard of. Symptoms include impaired speech and memory, trouble with attention and concentration, and even confusion. Fortunately, there are many ways to treat a thalamic stroke, and they may be worth exploring. However, to fully understand this condition, you must know the signs and symptoms. Listed below are some of the most common symptoms of a thalamic stroke.

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The majority of thalamic strokes occur in the right hemispheric, but there is no evidence to indicate that the right hemispheric is more common. This may be due to selection bias, as patients and next-of-kin tend to have more difficulty recognizing left-sided stroke symptoms. Furthermore, the symptoms of a thalamic stroke vary widely, depending on the area of the brain affected and the lateralization of the lesions.

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The earliest description of a thalamic stroke was published in 1906, when Dejerine e Roussy described speech and sensory motor disturbances related to the infarct in the lateral geniculate body. Today, thalamic stroke is classified into four main arterial territories. A thalamic infarct accounts for 11% of vertebral infarcts. It affects the posterior limb of the internal capsule, as well as the lateral geniculate, uncus, and cerebral peduncle. During the onset of the symptoms, the patient has decreased awareness and consciousness.

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Neuroimaging studies support this etiology. Although the underlying mechanism of thalamic stroke is not completely understood, the findings suggest that the thalamic tract is responsible for impaired language retrieval. In addition, the presence of bilateral SMA may play a crucial role in restoring language function after a thalamic stroke. While it is difficult to assess the specific location of damage in the thalamic lobe, this research shows that the SMA may be responsible for the word retrieval difficulty.

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The current study describes the clinical presentation of 24 patients with thalamic infarcts. The majority of patients in the study had inferolateral artery involvement. In one patient, the infarct affected the tuberothalamic artery. The other two patients had both inferolateral and paramedian arteries involved. While there are many other types of thalamic stroke, the study has highlighted a relatively rare type.

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Two arteries supply the thalamus: the posterior cerebral artery (P1) and the basilar artery. The posterior communicating artery (P2) provides blood to the ventrolateral nucleus. The ventroposterolateral nucleus receives inputs from the cerebellum and the medial lemniscal pathway. The lateral thalamus is supplied by the thalamogeniculate group of arteries.

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The thalamus is a critical component of sleep and plays a major role in regulating REM, slow-wave, and NREM states. Damage to the anterior nucleus of the thalamus may result in neuropsychological problems. In early involvement, patients may exhibit a decreased level of consciousness that may last hours or even days. They may also exhibit agitation, aggression, and decreased responsiveness.

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Patients with left thalamic lesions may exhibit an array of neuropsychological deficits. These include reduced verbal output, impaired comprehension, and hypophonic speech. Additionally, patients with right thalamic lesions may exhibit a disproportionately high incidence of pain syndromes. Left thalamic lesions may lead to an acalculia or aphasia. There are also several other types of thalamic lesions, including those in the posterior choroidal region.

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In addition, patients with an abnormal diffusion-weighted imaging (DWI) were screened for thalamic stroke seven days after admission to a hospital. DWI showed a pure thalamic infarct in four groups of the thalamic blood supply territory. These findings were consistent with a diagnosis of thalamic stroke. Following diagnosis, the patient underwent follow-up MRI scans, which revealed minute lesions.

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Patients with a thalamic stroke can experience acute movement disorders. The stroke disrupts important connections and fibres within the thalamus, and a delayed onset of these movements is possible. The recovery of movement following a thalamic stroke depends on the individual's individual neuroplasticity. Fortunately, if the thalamic infarct occurs in the hand, it can cause the hand to reappear and restore smooth, coordinated movements. Moreover, it can also cause aberrant connections that generate abnormal involuntary movements.

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