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  • Writer's pictureOren Zarif

Lacunar Stroke Symptoms - Oren Zarif - Lacunar Stroke


The earliest signs of lacunar stroke are usually temporary and can go away without treatment. These strokes are caused by small clots that interfere with blood flow. Because these clots can dissolve before they do much damage, patients may experience temporary symptoms that can improve within minutes. This type of stroke is also known as a transient ischemic attack and usually recovers completely within 24 hours. Symptoms may also be confused with a traumatic brain injury, such as a car accident.

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Another type of lacunar stroke involves the motor and sensory areas of the brain. Damage to the thalamus results in loss of sensory function and motor abnormalities. These symptoms occur on the same side of the body and can cause weakness or wobbly movements. In most cases, the face is not affected by lacunar stroke. In some rare cases, there may also be a postictal state that may be present.

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Although the symptoms of a lacunar stroke are relatively small and subtle, they are still very important. While the death of brain cells is an unpleasant experience, the symptoms of stroke will vary based on the location of the stroke. Lacunar stroke is often a silent killer, and the death of brain cells will disrupt bodily functions and prevent the patient from recognizing the signs of the disease. Symptoms may include mild cognitive impairment or early dementia.

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Lacunar stroke is an ischemic brain disorder caused by occlusion of a penetrating artery. They account for about one quarter of all cerebral infarctions. Lacunar stroke is characterized by an apparent paradoxical clinical course. The disease is characterized by favorable short-term outcome, but with significant mortality risk. It can be difficult to distinguish a lacunar stroke from other ischemic strokes due to the asymptomatic progression of small vessel disease.

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The arteries in the brain are very small and branch directly off of the large main artery. These arteries carry high blood pressure and are heavily muscled. High blood pressure can damage the lacunar arteries, which is directly responsible for a lacunar stroke. Likewise, high blood pressure can damage the walls of the arteries, causing atherosclerosis. As a result, patients may experience short-term and long-term disability.

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After a lacunar infarction, patients require care from a neurologist and a multidisciplinary team of professionals. This includes occupational therapists, physical therapists, and social workers. In addition, patients must continue with rehabilitation therapy, including physical therapy, and may be given muscle relaxants to help reduce the symptoms of spasticity. Patients should also be monitored regularly for any risk factors. While there is no one specific treatment, adherence to treatment guidelines can help patients avoid a second stroke.

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MRI and CTP have the potential to identify lacunar infarcts with high specificity, which may be particularly important in the case of this type of stroke. In a recent study, CTP demonstrated superiority in lacunar detection over NCCT/CTA and other MRIs, as indicated by a previous review of 1085 patients with acute stroke symptoms. Patients had CTP within 12 hours of a stroke episode and MRI within 7 days of the stroke onset. Infarcts of 2 cm or more were noted.

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