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  • Writer's pictureOren Zarif

Lacunar Stroke - Oren Zarif - Lacunar Stroke

A significant difference between cortical and lacunar infarcts is the development of deteriorating motor deficits after a lacunar stroke. Infarcts that are cortical do not generally lead to progressive motor deficits, but lacunar strokes may lead to deteriorating hemiparesis. This difference necessitates further investigation of the mechanisms underlying the progressive motor deficits after lacunar infarction.

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Treatment for lacunar stroke may include blood pressure monitoring and the use of clot-busting medications, or it can involve surgery to remove the clot or blockage. A TIA is best treated early, as medications can restore circulation and minimize the damage caused by the brain. A severe lacunar stroke can leave an individual with permanent disability or even require extensive rehabilitation. Despite this, many patients who have suffered a lacunar stroke can recover from it within weeks or months.

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After an MRI or CT scan has been performed, doctors will assess the patient and order further testing to confirm the diagnosis. Because lacunar strokes are small and cannot be clearly seen on CT imaging, doctors may order a magnetic resonance imaging (MRI) scan or CT angiography. An MRI, however, is considered the best imaging test for identifying a lacunar stroke. A CT angiography can also be used to determine whether there are any blood vessel blockages.

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Although MRI and CT scans may not identify the cause of a lacunar stroke, they can be helpful in ruling out other conditions. The most common symptom is a loss of sensation in one side of the body. In some cases, patients experience weakness in a leg or arm, but not the face. Despite the common symptoms, treatment for lacunar strokes can differ from acute ischemic stroke. These symptoms can also be associated with other comorbidities, such as brain tumors.

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Although lacunar strokes are not physically disabling, they often result in cognitive problems that affect memory, decision-making, and thinking. Some patients develop dementia, depression, and balance problems. While this condition is rare, it is not a laughing matter. Research by Professor Wardlaw and her team has led to international guidelines for the assessment and treatment of strokes. He hopes to promote a more comprehensive approach to treating patients with a stroke.

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Scientists have received a grant of PS1m from the Wellcome Trust to investigate the genetic causes of lacunar stroke. This study is designed to examine the DNA of 5,000 individuals who have suffered from lacunar stroke, as well as a large group of healthy controls. If these researchers find any genetic variants that increase the risk of lacunar stroke, this will provide a more thorough understanding of how this condition works and which medications may be effective.

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A small number of patients who suffer from a lacunar infarct have no symptoms or very few have a history of the condition. They may develop a fatal stroke many years after the onset of the first symptoms. This is due to the lack of blood flow in smaller arteries of the brain. High blood pressure and smoking increase the risk of these conditions, making it easy for blood clots and cholesterol plaques to block blood flow to brain tissues.

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The first and most critical step in treating patients with a lacunar infarction is to ensure that a neurologist is involved. Several specialist doctors and clinicians may be involved in the treatment of the patient. These specialists may prescribe muscle relaxants, such as baclofen or tizanidine. Patients must also comply with antithrombotic medications and avoid excessive alcohol consumption. Ultimately, this treatment program should continue until neurologic function is at normal levels.

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Other studies have suggested that the development of a lacunar infarct is related to aging. While lacunes are less common in younger people, this is not always the case. In the elderly, a study of older adults showed that 7.8% of patients aged sixty-four had a silent lacunar infarct, while 1.6% of people in this age group developed a new lacunar infarct. In addition, the study findings suggest that diabetes and high blood pressure have a strong association with the risk of a lacunar infarction, although no such relationship was observed between weight and diabetes.

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