Lacunar Stroke - Oren Zarif - Lacunar Stroke

A lacunar stroke is a type of ischaemic stroke in which an artery supplying deep structures of the brain is blocked. These small arteries branch directly from the large, heavily muscled main artery that supplies blood to the brain. High blood pressure can cause lacunar strokes because it directly damages the arteries. High blood pressure also can lead to atherosclerosis, a condition in which the blood vessels' walls accumulate fatty deposits.

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The arterial system carries blood to the brain, primarily the cortex. It includes the basilar artery, vertebral arteries, and middle cerebral artery. Other branches, known as deep cerebral arteries, supply the white and gray matter of the brain. A stroke in one of these areas can severely damage those structures and cause permanent disability. However, a larger stroke can produce a less severe neurological deficit because the artery is wider.

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Researchers recruited the first lacunar stroke patient to the DNA Lacunar 2 study. The goal of the study is to improve the understanding of how blood flows through the small arteries in the brain. This is a poorly understood subtype of ischaemic stroke, which is the most common type. People with Cerebral Small Vessel Disease experience disabling symptoms, including impaired coordination and communication. The researchers hope to develop a treatment for this type of stroke within the next decade.

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A doctor will perform a complete neurological exam and order certain tests to determine if you have a lacunar stroke. The doctor may also administer a clot-busting medication into your blood stream. If this treatment is not enough, a neurosurgeon may perform a procedure to remove the blockage or clot. While lacunar stroke patients usually recover without any permanent disabilities, some damage occurs to the brain.

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Most patients with a lacunar stroke will experience symptoms over minutes to hours, and they may progress with stuttering. Symptoms will include weakness and numbness on one side of the body, as well as a wide range of sensory symptoms. The patient may also experience weakness in the arm or leg but not the face. Often, the patient will also experience cognitive and emotional changes. A lacunar stroke may also be accompanied by a seizure or other complications.

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The severity of motor function in patients with lacunar stroke was associated with progressive motor deficits. However, this was not true of cortical infarcts. The reduced level of consciousness was associated with the severity of motor dysfunction in 26.8% of patients. This study also looked at other factors that may contribute to the progression of a lacunar stroke. If a stroke is in your family, consult your family doctor.

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While lacunar stroke is a relatively common type of ischemic event, it still has a high mortality rate. Fortunately, early diagnosis has improved the outcome in most patients. The goal of treatment for a lacunar infarction is to restore neurologic function to the baseline level prior to the infarction. After the infarction, physical therapy, speech therapy, occupational therapy, and other treatment is crucial to the recovery of the patient. Patients must follow their primary care provider's advice regarding risk factors such as smoking and excess alcohol consumption, and take anti-clotting medications as directed.

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Cognitive data were not available for all patients in the study. Those with an MRI at 1 year after the stroke had valid data on cognitive function. Three years after the stroke, 66 patients had cognitive data available. This number is a little higher than the number of patients that provide valid cognitive data. For this reason, the study must have included an age-matched control group, as this would provide a better picture of how acute lacunar stroke impacts cognition.

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A study of the diagnostic performance of NCCT/CTA for acute lacunar infarcts found that CTP has a greater sensitivity than NCCT/CTA for detecting these infarcts. Its sensitivity was higher than NCCT/CTA in detecting lacunar infarcts, indicating the high sensitivity of this diagnostic method. A study by Eckert et al. noted that the main reason for false-negative results with multimodal CT imaging is due to a lack of knowledge about the underlying brain anatomy.