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  • Writer's pictureOren Zarif

Lacunar Stroke - Oren Zarif - Lacunar Stroke

A quarter of ischaemic strokes are lacunar. The cause is unclear, but a possible risk factor is fatty deposits in the bloodstream. These deposits can cause blockages in the arteries, causing them to thicken and infarct. The condition is a result of a stroke, which can lead to other complications. The researchers hope to develop a treatment in the next 10 years. But for now, the only effective treatment is observation.

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The study also found that both groups of patients had intracranial and extracranial vascular disease. However, the degree of carotid atherosclerosis varied between the two groups. Patients in group A exhibited more nonstenotic plaques and low-grade stenoses than patients in group B. The risk of a lacunar stroke is greater in those with a moderate-to-severe ICA.

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The progression of lacunar stroke depends on the part of the brain that is deprived of blood. Different areas of the brain are responsible for different functions, including movement, sensation, speech, and balance. Patients with prolonged high blood pressure may develop more than one lacunar stroke. These strokes can lead to mental and emotional issues, and in some cases even dementia. The symptoms may vary according to the region of the brain affected, but there are some common signs.

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Early treatment is essential for a person to recover from a lacunar stroke. Treatment usually involves antihypertensive therapy, high-dose statins, and close monitoring of blood glucose levels. Patients may also need outpatient rehabilitation. If there is no other risk factor for lacunar stroke, the chances of recovering from it are high. The recovery process depends on the extent of damage and the extent of disability. But the sooner treatment is sought, the better the chance for recovery.

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Although the symptoms of lacunar stroke can vary, treatment is critical and must be given as soon as possible. Acute treatment can help patients recover quickly, often within hours. In fact, 90 percent of patients with a lacunar stroke will recover substantially within three months. It is important to treat a lacunar stroke immediately, as this type of stroke may require physical rehabilitation and may result in permanent disability. So, treatment for lacunar stroke is vital if it is suspected.

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Lacunar stroke is typically caused by damage to the thalamus, which controls motor and sensory functions. Usually, a stroke in this area will cause weakness of the arm or leg, while a pure sensory lacunar stroke will lead to sensory abnormalities. This type of stroke usually affects the thalamus, which is heavily involved in processing all senses. The stroke will affect the side opposite to the affected side.

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Research on lacunar stroke should aim to increase the sample size and follow-up period. These factors will improve statistical power. One solution to the sample size issue could be to use an international database, such as a branch of the PLORAS database or the Meta VCI Map consortium. Because different studies use different cognitive tests, comparisons of these results may be difficult. Therefore, it is imperative to consider all potential risk factors when selecting treatment.

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Small noncortical infarcts arising from the lenticulostriate branch of a cerebral artery may result in lacunar stroke. Infarcts in this region usually do not cause significant disability, but a repeated episode may lead to vascular dementia. The condition can also be asymptomatic. This condition has been associated with several risk factors, including diabetes mellitus and hypertension.

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Other symptoms include ataxic hemiparesis, dysarthria, clumsiness, and facial weakness. In addition, the patient may display ipsilateral hyperreflexia and the Babinski sign, which indicate that the brain is infarcted in the thalamus. Dysarthria-clumsy hand syndrome is the least common lacunar syndrome and may involve weakness of the hands and facial muscles.

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In the LACI+/+ group, there was a longer time interval between the first stroke and recurrence. This interval was 495 days, compared to 340 days for those in the LACI-/-group. The longer the patient survived after a stroke, the greater the risk of developing another stroke. These findings offer hope for the future. With continued research, stroke prevention can be achieved. Once again, these new treatments are available, they will help millions of people live healthy and happy lives.

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