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  • Writer's pictureOren Zarif

Lacunar Stroke - Oren Zarif - Lacunar Stroke

A small subtype of stroke, lacunar stroke, accounts for about 25 percent of all ischaemic strokes. It is often associated with limb weakness, speech difficulties, and paralysis. Researchers have been trying to understand its underlying causes. They believe it is caused by a small perforating artery located in the white matter of the brain. Currently, it is believed to occur in the corona radiate, internal capsule, and subcortical white matter.

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The prevalence of lacunar stroke is also increasing with age. However, the prevalence of lacunes varies among people of different racial and ethnic backgrounds. Although the occurrence is much lower among African Americans than whites, it may still be a significant cause of death in these communities. The researchers conducted a study to determine how many of these strokes occur in different groups and age ranges. They found that the incidence of lacunar stroke was higher in older people than in younger people.

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The symptoms of lacunar stroke may be mild or severe. A doctor will perform a detailed neurological examination to determine the extent of brain damage. A specialist may also administer medications directly into the brain to reduce the risk of a second stroke. A patient with lacunar stroke should seek medical attention as soon as possible. This type of stroke can have devastating effects, even permanently limiting the person's ability to function. Luckily, if it is diagnosed early, a person can recover.

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The signs of lacunar stroke depend on which part of the brain has been affected. The damage to the thalamus or internal capsule will cause sensory loss. In severe cases, the resulting motor abnormalities may cause weakness in one side of the body. In severe cases, the patient may have a wobbly gait, a stuttering gait, or even dementia. Although a patient with a lacunar stroke will most likely experience one of these symptoms, the majority of patients will be unable to move or communicate.

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Post-stroke, lacunar infarction requires intensive treatment by a multidisciplinary team. Physical therapy and occupational therapy are crucial for the patient's recovery. Medications used to treat spasticity may also be prescribed. A patient must follow all guidelines to prevent a recurrence of lacunar stroke by limiting risk factors, such as alcohol and smoking. Patients with these risks should follow a strict regimen of medication and lifestyle changes.

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Another weakness of the study was that there was no comparison group. Although lacunar stroke is a common cause of death, the causes of cognitive decline are different in the two subtypes. Some researchers believe that the same mechanism may underlie both types. It is important to understand the difference between lacunar stroke and other subtypes of stroke, as this may influence the prevention and treatment. The current data on lacunar stroke are limited, but the potential for cognitive decline remains.

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Another potential cause of lacunar stroke is a deep penetrating branch occlusion. Micro atheromas in the lenticulostriate artery can occur as well as macroatheromas caused by parent artery occlusion. Other causes of small cerebral infarcts include vasculitis, embolism, and infections. Despite the fact that autopsy cannot prove this, atherosclerosis of small cerebral arteries can also cause lacunar stroke.

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In the event of a lacunar stroke, the ischemic brain is most commonly affected by the death of brain cells. These deaths are often long after the stroke. Autopsy material from lacunar infarcts is sparse, as small cerebral vessels require meticulous dissection. The majority of studies, however, use a clinical diagnosis of stroke type in the hope of determining risk factors. It is vital to identify the cause of any lacunar stroke before it causes permanent damage.

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Various neuroimaging techniques can help diagnose a lacunar stroke. Currently, the most sensitive technique is CTP. This imaging technique has a high specificity for lacunar stroke, with sensitivity ranging from 85 percent to 93 percent. NCCT/CTA, on the other hand, does not reliably detect lacunar infarcts. For these reasons, it is not recommended for patients with a suspected lacunar stroke.

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