• Oren Zarif

Clinical Manifestations of Lacunar Lesion in Individuals With Silent Lacunes - Oren Zarif - Lacunar


The most common type of lacunar lesion is one in which the brain shows a single, silent lacune. However, there are also cases of multiple lacunes. In the present study, we will look at the clinical manifestations of lacunar lesion in individuals with silent lacunes. The main findings of this study are as follows:

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Symptoms of lacunar stroke are not as serious as those of full-blown stroke. If treated promptly, it can result in a partial or full recovery. Treatment is typically immediate with medicines to restore circulation. However, if the symptoms are severe, it could require a lengthy recovery, requiring physical rehabilitation and even permanent disability. To treat a lacunar stroke, you need to be evaluated by a physician immediately. Fortunately, most patients recover substantially within three months.

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First, doctors will examine the patient's symptoms and medical history. They may order an electrocardiogram. A detailed neurological exam will reveal any damage to brain functions. Further tests may reveal if there are any blocked blood vessels in the brain. Depending on the severity, a lacunar stroke may require heart function tests. Treatment may include the use of aspirin or other medications to reduce the risk of a subsequent stroke. If a stroke does occur, supportive measures may also be required, such as a mechanical ventilator or a heart-lung machine.

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Among the risk factors for lacunar stroke are advanced age, hypertension, smoking, diabetes mellitus, and elevated cholesterol. Prior stroke is a risk factor. Other risk factors include high cholesterol and obesity, as well as alcohol and drug use. However, some studies have shown that a significant portion of lacunar strokes are caused by embolic sources. The risk of subcortical dementia is also increased. So, if you are experiencing one of these strokes, don't hesitate to seek medical attention and get the treatment you need.

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Symptoms of a lacunar stroke depend on which part of the brain has been deprived of blood supply. Different parts of the brain are responsible for different functions, including movement, sight, speech, and balance. If you have suffered a long-term case of untreated high blood pressure, you may experience multiple lacunar strokes. The latter can result in emotional behaviors and dementia. However, these symptoms may be mild and temporary.

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The mechanism of lacunes is still unclear. It is thought that emboli from larger arteries or the heart may contribute to lacunes. However, cross-sectional observational studies are inconclusive in establishing causality. Other studies have only focused on patients who have symptoms of lacunar stroke. If we can identify the cause of lacunar lesion, then it will help us to develop effective secondary prevention regimens.

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MRI-defined lacunar infarcts are not rare in healthy elderly people. Although age, diastolic blood pressure, creatinine, and lipid levels were significant risk factors, these differences are not substantial enough to suggest a different underlying mechanism. Aside from MRI-defined lacunes, patients with silent lacunes are also at increased risk for stroke in the upper and lower extremities. However, further studies are needed to determine their role in predicting subsequent risk of stroke.

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Acute ischemic stroke can cause significant damage to the brain. It can result in a permanent disability or even a subsequent stroke. Therefore, it is imperative to seek medical attention if you suspect you're experiencing a lacunar stroke. Early diagnosis and treatment are the only surefire ways to prevent irreversible brain damage. A lack of proper blood flow prevents brain cells from receiving proper nutrients. While the symptoms of lacunar stroke vary from one person to another, it is crucial to seek medical attention as soon as possible.

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Small, non-cortical infarcts in the brain are known as lacunar infarcts. These infarcts typically involve one small penetrating artery. These infarcts can result from embolism, lipohyalinosis, or even vasospasm, but there is no evidence to support this theory. This type of stroke is associated with ill-defined hypodensities and can occur in either chronic or acute forms.

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A significant difference between a WML and a lacunar infarct is its size. Lacunar infarcts are smaller in diameter than their corresponding cortical counterparts, but they are confluent and may cause cognitive problems in patients. Both types are classified as ischaemic strokes, but the latter is the most complicated. This type of stroke can lead to cognitive decline and co-ordination problems.

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