Causes and Symptoms of Wallenberg Syndrome - Oren Zarif - Wallenberg Syndrome
The cause of Wallenberg syndrome is not yet well understood. While some researchers have linked heart disease and artery disease to the condition, others have not found any definitive link between these conditions. Minor trauma to the neck is a leading cause of the syndrome in younger individuals. Patients may also experience pain, loss of sensation, and tinnitus. Treatments for Wallenberg syndrome include early physical and occupational therapy and speech therapy. A thorough clinical examination is necessary to determine if the condition is the cause of your child's symptoms.
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One case report of a patient with lateral medullary syndrome describes a 30-year-old right-handed female with a recurrent headache, vertigo, and weakness in her extremities. She underwent neurologic testing and MRI, which revealed lingual dysarthria, right dysmetria, and nystagmus. Magnetic resonance imaging revealed a large left lateral medullary infarction with involvement of the left middle cerebellar peduncle. The patient recovered and was discharged from the hospital three weeks later. She later recovered in a rehabilitation facility.
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Symptoms of Wallenberg syndrome may range from mild dizziness and vertigo to full-blown stroke. Patients may experience difficulty walking, standing, or sitting, and their voice may be hoarse or weak. Although complete Wallenberg syndrome is rare, partial Wallenberg syndrome is often sufficient for diagnosis. In most cases, the condition can be corrected with surgery. The condition is more likely to improve if the patient is able to undergo MRI.
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The most common cause of Wallenberg syndrome is ischemic stroke, which occurs in the posterior circulation of the brain. Most cases are triggered by a thrombus or embolism. Less common causes include mechanical trauma to the vertebral artery of the neck, arteriovenous malformations, and multiple sclerosis. When it is associated with ischemic stroke, MRI can be normal. However, MRI may be normal in up to 30% of patients.
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The overall prognosis of people with Wallenberg syndrome is improved compared to those with other acute ischemic strokes. The majority of patients with this disorder recover well, but lingering problems can affect walking or balance. Early physical therapy and occupational therapy is crucial for recovery. The symptomatic symptoms of the condition include gait instability, ataxia, and hiccups. However, treatment depends on the severity of the disease and the size of the infarct.
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The most important factor in the diagnosis of Wallenberg syndrome is the location of the infarct in the posterior inferior cerebellar artery. In 1894, Wallenberg performed a post-mortem on a patient suffering from this condition. His findings indicated that the infarct was located in the posterior inferior cerebellar artery. If not treated, this condition can lead to lateral medullary infarction.
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The main goal of treatment for WS is to improve visual symptoms. Depending on the cause, treatment for WS can be time-dependent. Intravenous tissue-type plasminogen activator (TPA) can be given in a matter of hours. The patient can return to oral feeding within a month to two. This can be a difficult procedure to perform, but the patient is largely expected to improve. It's important to be aware of the symptoms of WS to determine what treatment is best for you.
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The severity of the condition will depend on the severity of the symptoms, as well as the underlying cause. Treatment should aim to improve a patient's quality of life and help him or her regain the function he or she lost. In addition to conventional medications, patients may undergo a variety of therapies. In addition to speech therapy, swallowing exercises can also be very helpful for patients with WS. Besides speech therapy, specialized neuromuscular electrical stimulation is used in certain cases. A speech and language pathologist or an occupational therapist would normally administer VitalStim.
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During swallowing, electrophysiological studies have revealed that patients with WS experience more oral than pharyngeal dysphagia than other patients. In patients with WS, laryngeal elevation may also be delayed compared to other hemispheric stroke victims. In some cases, the contralateral center of the medulla oblongata is responsible for overcome dysphagia.